|
Please
download and print the PDF file below:
|
|
|
|
Maureen
Michael, BSN, MBA, CHN
|
Maureen Michael, BSN, MBA, CHN, is
Executive Director of Central Florida Kidney
Centers, Inc., Orlando, FL, and a member of the
NKF-K/DOQI Steering Committee. She is a
member of ANNA's Sunshine Central Chapter.
|
|
Donna Garcia, RD, LD/N |
Donna Garcia, RD, LD/N, is Renal Dietitian,
Central Florida Kidney Centers, Inc., Orlando, FL.
|
Secondary hyperparathyroidism, a characteristic manifestation of chronic kidney disease
(CKD), arises from a series of abnormalities in the interrelated cascade that controls bone
and mineral metabolism. Although bone disease is the most recognized consequence of secondary
hyperparathyroidism (HPT), it also precipitates a series of potentially devastating
effects on the cardiovascular, nervous, endocrine, and erythropoietic systems. Optimal management
of secondary hyperparathyroidism should allow control of serum parathyroid hormone
levels while preventing hyperphosphatemia, maintaining normal calcium levels, and
providing adequate vitamin D supplementation. Current therapeutic approaches are frequently
unsuccessful in achieving these goals and may, in fact, exacerbate risk factors that
increase morbidity and mortality in the dialysis population. Development of new therapeutic
approaches may provide improved control of secondary hyperparathyroidism in the future.
|
|
CE
RELATED INFORMATION |
| This
offering for 1.8 contact hours is being
provided by the American
Nephrology Nurses Association (ANNA),
which is accredited as a provider and approver
of continuing education in nursing by the American
Nurses Credentialing
Center-Commission on Accreditation (ANCCCOA). This educational
activity is approved by most states and specialty
organizations that recognize the ANCC-COA accreditation
process. ANNA is an approved provider of continuing
education in nursing by the California Board of
Registered Nursing, BRN Provider No. 00910.
|
|
Goal:
To discuss the effects of secondary hyperparathyroidism (HPT) on
the entire body.
Objectives:
1. List the potential adverse clinical outcomes related to inadequately controlled parathyroid
hormone levels.
2. Describe the pathophysiology of secondary hyperparathyroidsim.
3. Discuss current therapeutic interventions and newer therapeutic approaches to treatment of
secondary hyperparathyroidism.
|
AUTHOR DISCLOSURE
STATEMENT |
Maureen
Michael has no conflicts of interest to disclose.
Donna Garcia has no conflicts of interest to
disclose.
|
ANNA and HDCN CE POLICY STATEMENTS |
The CE policy and disclosure statements of the American Nephrology
Nurses' Association are given in detail on the Symposium
Home Page. The CE policy statements of HDCN are listed on
this page. |