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Article Review/Hyperlink
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Merkel PA, Chang Y, Pierangeli SS, Convery K, Harris EN,
Polisson RP
Prevalence and clinical associations of anti-cardiolipin
antibodies in a large inception cohort of patients with
connective tissue diseases
Am J Med
(Dec) 101:576-583 1996

Hypertension is the second most common cause of end-stage renal disease in
the U.S. overall and the most common cause in African-Americans. The
relative incidence of new ESRD caused by hypertension in AAs is 6-fold
higher than non AAs. There are probably many reasons for this rather large
discrepancy in rates of disease including higher prevalence of
hypertension, more severe hypertension, access to better medical care and
others. However, the entire difference cannot be accounted for on the
basis of known risk factors. Moreover, the proof that ESRD is actually
caused by hypertension in any population has not been forthcoming because
very few individuals undergo renal biopsy.
In this study 46 of 95 participants in the AASK study underwent renal
biopsy either prior to or at the time of enrollment into baseline of the
pilot study. Study entry criteria included hypertension (seated diastolic
> 95 mmHg), age range 18-70 and a GFR range of 2-70 ml/min/1.73 m2.
Patients with diabetes and heavy urine protein/creatinine ratio > 2.5
were
excluded. Biopsy material was subjected to light, immunofluorescence and
electron microscopy. Adequate biopsies were available in 39 of the 46
participants and 38 of those showed arteriosclerosis and/or
arteriolosclerosis. Segmental sclerosis was present in 5 of these cases
and 1 patient had clinical findings suggesting focal segmental
glomerulosclerosis. Interestingly, global glomerulosclerosis was present
in 43% of cases and this lesion correlated positively with systolic blood
pressure, serum cholesterol, reciprocal of serum creatinine and
interstitial fibrosis.
The authors concluded that renal biopsies in AA with mild to moderate
renal insufficiency in the absence of marked proteinuria are overwhelmingly
likely to reveal vascular lesions consistent with hypertensive
nephrosclerosis.
Comment: This is an important study for several reasons. First, the
demographic characteristics of this study population match those of
published at high risk populations for ESRD attributed to hypertension
indicating that this is the target population sought for in the AASK study.
Second, the kidney biopsy in this situation strongly supports the clinical
diagnosis of hypertensive nephrosclerosis. Third, it possible that there
is an important pathogenetic link between the global glomerulosclerosis and
systolic hypertension and perhaps hypercholesterolemia.
These data may help to find additional markers that indicate increased
risk of progressive renal disease and perhaps point a way towards better
therapies that can reduce risk or halt disease progression in the future.
(Robert D. Toto, M.D., University of Texas Southwestern
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