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Article Review/Hyperlink
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Pagtalunan ME, Miller PL, Jumping-Eagle S, Nelson RG, Myers
BD, Rennke HG, Coplon NS, Sun L, Meyer T
Podocyte loss and progressive glomerular injury in type II
diabetes
J Clin Invest
(Jan) 99:342-348 1997

The occurrence of diabetes mellitus (type II) and consequent
nephropathy in Pima Indians at an earlier age compared to other ethnic
groups provides an opportunity to study renal morphologic
abnormalities with little interference from age related changes.
In this study the authors biopsied 51 Pima Indians who belonged to
four groups: early diabetes without albuminuria, long term diabetes
without and with microalbuminuria, and clinical nephropathy. The
control group consisted of eight non-Pima Indians who were kidney
donors. There was a progressive increase in glomerular volume,
fractional mesangial volume and GBM thickness compared to controls.
The width of the foot processes increased in diabetic patients over
time. The actual number of podocytes per glomerular tuft was decreased
in patients with clinical nephropathy. However, the density of
podocytes in the glomerualr tuft was progressively lower and the area
occupied by the individual podocyte, podocyte volume and number of
nonpodocyte cells per glomerular tuft were progressively higher with
passage of time. The authors suggest that podocyte loss contributes to
the progression of diabetic nephropathy.
Comment: This article addresses the role of podocyte in
diabetic nephropathy, an area that has not received much attention in
the past. It is conceivable that decrease in podocyte population
results in stretching of the remaining cells as the glomerular volume
expands. This act of stretching may cause important change in the
cytoskeleton and affect the function of the cell, eg., synthesis of
matrix components. A reduction in the actual number of cells per
glomerulus would also raise the possibility of apoptosis as a
mechanism of podocyte loss. The major issue of concern in this paper
is the lack of appropriate controls. The morphometric parameters in
Pima diabetics are compared to non-Pima Indian controls. It is known
that in normal nondiabetic Pimas the glomerular volumes are twice that
seen in whites (Schmidt K et al, J Amer Soc Nephrol 3:229,
1992). Schmidt and associates could not find any increase in
glomerular volume in established diabetic nephropathy among Pima
Indians (ibid). Therefore, it is unclear if a reduction in podocyte
density actually occurs relative to controls. Additionally, the
findings of decrease in podocyte density and loss of renal function is
correlative. A cause and effect relationship remains to be shown.
However, the paper has rekindled interest in potential role of the
podocyte in diabetic nephropathy.
(B.S. Kasinath, M.D., University of Texas at San Antonio)
The full text of this article is available from the
JCI online site at
this link
.
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