Drug Alert: Cisapride/cytochrome P450


Cisapride (Propulsid®) October 14, 1995
A drug alert ("important safety information") was issued by Janssen Pharmaceuticals on the above date, that "serious cardiac arrhythmias, including ventricular tachycardia, ventricular fibrillation, torsades de pointes, and QT prolongation have been reported in patients taking PROPULSID® with other drugs that inhibit cytochrome P450 3A4, such as ketoconazole, itraconazole, miconazole, troleandomycin, erythromycin, fluconazole, and clarithromycin. Some of these events have been fatal. PROPULSID® is contraindicated in patients taking any of these drugs." For further information contact Janssen at Washington Crossing, 1125 Trenton-Harbourton Road, Post Office Box 200, Titusville, New Jersey 08560-0200.

Comments:
Dr. George Aronoff (Louisville, KY)
(Send questions and suggestions to: graron01@ulkyvm.louisville.edu):

The important safety information issued by Janssen Pharmaceuticals for cisapride is the latest in a series of alerts regarding drugs which influence the cytochrome P-450 metabolizing system. Cytochrome P-450 is a superfamily of more than 100 enzymes catalyzing a large number of bioactivation and detoxication reactions. The large number of different cytochrome enzymes and genetic polymorphism in the activity of these enzymes complicates the use of drugs metabolized by them.

Cytochrome P-450 IIIA, one of the cytochrome enzymes, is inhibited by ketoconazole, itraconazole, miconazole, troleandomycin, erythromycin, fluconazole, and clarithromycin. Drugs metabolized by this enzyme, like cisapride, may accumulate and cause toxicity when taken concurrently with drugs that inhibit cytochrome P-450 metabolism. Serious arrhythmias characterized by QT prolongation, including ventricular tachycardia, ventricular fibrillation, and torsades de pointes have been reported. This mechanism is the same as that previously reported for the interaction between terfenidine (Seldane) and erythromycin.

Cyclosporine: Cytochrome P-450IIIA is also primarily responsible for cyclosporine metabolism. Other drugs known to inhibit cytochrome P-450IIIA include verapamil, diltiazem, nicardipine, nifedipine, and nitrendipine, theophylline terfenadine and astemizole. Grapefruit juice inhibits cytochrome P-450IIIA and can enhance the absorption of cyclosporine. Concurrent use of these drugs or the ingestion of grapefruit juice may substantially increase cyclosporine plasma concentrations. Although toxicity is the major concern, ketoconazole or grapefruit juice have been used to purposely increase cyclosporine plasma concentrations in an attempt to decrease the dose and expense of cyclosporine.

Other cytochrome P-450 enzymes:Several other cytochrome P-450 isoenzymes have been well characterized. They include cytochrome P-450IID6, known to metabolize the antiarrhythmic agents encainide, flecainide, and propafenone, the beta blockers propranolol, metoprolol, and timolol, the antidepressants fluoxetine, paroxetine, fluoxamine, imipramine, desipramine, amitriptyline, and nortriptyline, as well as codeine. Cytochrome P-450IIC metabolizes the following drugs the anticonvulsants phenytoin, mephenytoin, and methobarbital, oral hypoglycemic, tolbutamide, and warfarin.

Because of the many drug interactions effecting the cytochrome P-450 family of enzymes, caution should be used when administering the many drugs metabolized by these enzymes. Establishing specific diagnoses before drug prescribing, using as few drugs as possible, and monitoring for efficacy and toxicity are useful in decreasing adverse drug events.

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