Mallamaci F, Tripepi G, Zoccali C
Vasodepressor syncope in haemodialysis patients
16th Scientific Meeting of the International Society of Hypertension
ISH Abstract Book (Jun) 16: 1996

Hypotension is the main complication of haemodialysis treatment. It has recently been shown (JCI, 90, 1657, 1992) that it may be caused by the activation of a cardiovascular reflex causing abrupt sympathetic withdrawal, vasodilatation and bradycardia. This study is aimed at assessing the prevalence of vasodepressor syncope (defined as hypotension associated with bradycardia) in a large dialysis population (n = 106) and test the hypothesis that dialysis patients are predisposed to vasodepressor syncope.

Twenty patients with well defined dialysis hypotension (i.e. MAP fall > 20% in > 80% of dialysis treatments) were prospectively identified during a 3-months survey. These cases underwent non invasive haemodynamic monitoring and TBW measurement during a standard haemodialysis session. In addition 18 dialysis patients, including 7 patients with dialysis hypotension underwent a tilt test in a volume-depleted state (after dialysis).

Sixty hypotensive episodes were recorded. Heart rate rose in 35 episodes, did not change in 19 episodes and fell in 6 episodes. The HR response pattern to hypotension was highly reproducible in 10 patients (always tachycardia: 6, always unchanged heart rate: 4). Patients developing bradycardic hypotension (n = 5) all had an erratic HR response to hypotension (i.e. bradycardia preceded or followed by tachycardia or by no HR change) and were characterized either by the typical haemodynamic pattern of hypovolemia (predialysis hypotension, tachycardia and low TBW) or by being treated with a very high UF rate (greater than 0.36 ml/Kg/min). On tilt testing 4 patients developed bradycardic hypotension (two hypotension-prone and two hypotension-resistant dialysis patients).

The overall proportion (20%) of tilt induced vasodepressor syncope in uraemic patients did not differ from that reported in healthy subjects and in non-uraemic patients (Kapoor, Am J Med 1994, 97, 75). Tachycardia is the more frequent HR response pattern to hypotension in dialysis patients. Vasodepressor syncope in dialysis patients is consistently associated with marked hypovolemia.

Comment: This study adds weight to the hypothesis that bradycardic hypotension in dialysis patients is a physiological response to severe hypovolemia rather than the expression of a peculiar predisposition to vasodepressor syncope (Carmine Zoccali, M.D, Reggio Calabria, Italy).

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16th Scientific Meeting of the International Society of Hypertension
Basic hemodialysis : Complications (acute)